Research Paper on Impact of Painkillers In The Urinary Tract Infections (UTI)

Introduction

According to Koskimaki et al. (2001) over the last decade, researchers have confirmed that a significant global population has been infected with Urinary Tract Infections. The latter has motivated medic scholars from to Washington University to seek immediate intervention toward the approach (Koskimaki et al., 2001). The infection is caused by a bacterium known as Escherichia coli which inhabit the bowel. The research concluded that the immune protein induces inflammation (COX-2). Consequently, NSAIDs like ibuprofen blocks protein causing inflammation, thus, supporting the impact of painkillers in UTIs.

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Male Reproductive System

A male reproductive system avail range of functions including provision of hormones such as testosterone which endorses the sexual wellbeing of males and ensures fertility (Roehrborn, 2004). In most cases, some problems that arise are often treatable while others potentially pose critical consequences. For instance, Benign Prostatic Hypertrophy (BPH) which is common in older men and involves enlargement of prostate gland. The male with this condition then experiences a low stream of urine and strains during urinating. It is the cause of lack of proper understanding, though relieved via surgery or medication in case of symptoms persist.

Effects of Deep Exercises On The Female Reproductive System

Recently, women have increasingly become physically active. However, despite exercises providing substantial benefits on health, excessive exercise is linked to the unique set of risks on the female athletes (Warren & Perlroth, 2001). The condition is referred to as Hypothalamic dysfunction. It results from strenuous exercise. In which causes disturbance of GnRH pulsatility, resulting menarche delay, hence disrupting menstrual cycle. Precise mechanisms trigger variance in reproductive dysfunction across athletic disciplines. The condition is treated with exogenous estrogen to stimulate hormonal balance. Notably, the treatment aims to correct metabolic abnormalities through remedying estrogen deficiencies.

Lymphatic System: Linkage of Metabolic and Inflammation Systems.

Metabolic Syndrome (MS) and associated with dietary changes pose a significant threat to the modern population due to their unhealthy eating habits. Existing inflammatory conditions are likely to prone the victim to MS and other cardiovascular diseases (Chakraborty, Zawieja, Wang, Zawieja, & Muthuchamy, 2010). The same is supported by a decrease in the level of proactive adiponectin and adipokine which further emphasize the connections between MS such as obesity and body Inflammation.

Cardiovascular System

This can be described as the most critical system in human anatomy as it is responsible for the supply of blood throughout the body. The heart is the primary organ in the system suffers many conditions termed as heart and blood vessel disease (AHA Editors, 2017). These conditions result when the flow of blood is impaired, in processes such as atherosclerosis. Clots that form in blood vessels narrow them making blood flow difficult and increases blood pressure. Clots also result in heart attacks when the heart itself fails as a result of blood not flowing to the heart. Heart failure occurs at varying degrees. The condition implies that the heart is not functioning normally (AHA Editors, 2017). All these cardiovascular conditions can be prevented and treated through proper diet, exercise and medication.

References

AHA Editors. (2017, June 7) What is cardiovascular disease? Retrieved from American Heart Association: http://www.heart.org/HEARTORG/Conditions/What-is-Cardiovascular-Disease_UCM_301852_Article.jsp#.Wqg3veeYNEY
Chakraborty, S., Zawieja, S., Wang, W., Zawieja, D. C., & Muthuchamy, M. (2010). Lymphatic system: a vital link between metabolic syndrome and inflammation. Annals of the New York Academy of Sciences, 1207(1).

Koskimaki, M. Hakama, H. Huhtala, TLJ Tammela, J. (2001). Association of non-urological diseases with lower urinary tract symptoms. Scandinavian journal of urology and nephrology, 35(5), 377-381.

Roehrborn, C. G. (2004). Benign prostatic hyperplasia. Textbook of Benign Prostatic Hyperplasia, 295.

Warren, M., & Perlroth, N. (2001). The effects of intense exercise on the female reproductive system. Journal of Endocrinology, 170(1), 3-11.